"One theory is that the change in caloric intake beefs up cell repair mechanisms against destructive agents called free radicals."
Since 1962 we have been told that cancer is a nutritional disease. Now we have the added and cumulative effect of environment, including vaccines. Even veterinarians are relating cancer in companion animals to the vaccines.
The use of the term "free radicals" also tells you that there is a very real reason why sound nutrition and vitamins are helpful in the process of recovering from cancer.
Articles like this make me wonder if any of the so called researchers know what they are doing, or even what they are saying.
If I look at just one cancer drug, 5 FU, a fluoride based long used chemo drug, I find that it depletes these nutrients -
NB:Limited test tube research occasionally does support the idea that an antioxidant can interfere with oxidative damage to cancer cells. However, most scientific research does not support this supposition.
Multiple nutrients* (malabsorption)
Taurine* reduces the likelihood and/or severity of a potential side effect caused by the medication.
Beta-carotene* (mouth sores)
Chamomile* (mouth sores)
Eleuthero* (Eleuthero (Eleutherococcus senticosus)
Russian research has looked at using eleuthero with chemotherapy. One study of patients with melanoma found that chemotherapy was less toxic when eleuthero was given simultaneously. Similarly, women with inoperable breast cancer given eleuthero were reported to tolerate more chemotherapy.45 Eleuthero treatment was also associated with improved immune function in women with breast cancer treated with chemotherapy and radiation.)
Glutamine* (mouth sores)(intestinal toxicity)
N-acetyl cysteine* (NAC)
Probiotics (Lactobacillus GG)
Spleen peptide extract* (see text)
Thymus peptides* (see text)
Vitamin E*, topical (mouth sores)(also prevents hair loss)
PSK* (Coriolus versicolor)- The mushroom Coriolus versicolor contains an immune-stimulating substance called polysaccharide krestin, or PSK. PSK has been shown in several studies to help cancer patients undergoing chemotherapy. One study involved women with estrogen receptor-negative breast cancer. PSK combined with chemotherapy significantly prolonged survival time compared with chemotherapy alone. Another study followed women with breast cancer who were given chemotherapy with or without PSK. The PSK-plus-chemotherapy group had a 25% better chance of survival after ten years compared with those taking chemotherapy without PSK. Another study investigated people who had surgically removed colon cancer. They were given chemotherapy with or without PSK. Those given PSK had a longer disease-free period and longer survival time. Three grams of PSK were taken orally each day in these studies.)
New clue on why cancer spread is linked to diet Mar 11, 2009
PARIS (AFP) – Investigators on Wednesday said there could be a clue to explain why dietary restriction succeeds or fails in braking the growth of cancer.
Scientists spotted a century ago the link between calorie intake and the progression of certain kinds of tumour.
But how this works at the molecular level has been unclear. In addition, tumours may remain puzzlingly immune to fasting among some individuals but not among others, which is partly why the practice has not been incorporated into cancer treatment.
Nada Kalaany and David Sabatini of the Massachusetts Institute of Technology (MIT) looked at the impact of food curbs on six types of human cancers that had been grafted onto mice which had been genetically modified to lack immune resistance.
The mice were then split into two groups, with one batch able to tuck into a standard rodent diet for two to three weeks. The others received daily meals that amounted to a 40 percent reduction in calories compared to their counterparts, which led to a fall of between 20 and 30 percent in body weight.
The researchers conclude that tumours' sensitivity to fasting depends on a key pathway -- activation of an enzyme called phosphatidylinositol-3-kinase, also called PI3K, which plays an important role in the proliferation of cancer cells.
If the PI3K pathway is closed, tumours respond to dietary restriction, and vice versa.
The study is important because it helps drug designers who are working hard on prototype treatments that mimic the effects of dietary curbs on cancer.
As several cancer types carry mutations that could cause the PI3K mechanism to open up, the paper suggests that the approach will not work in certain cases.
The paper is published by the British-based journal Nature.
Previous research on lab rodents suggests that a calorie decrease of 10 to 50 percent leads to a reduction in the incidence of certain cancers and brakes the speed at which they spread.
One theory is that the change in caloric intake beefs up cell repair mechanisms against destructive agents called free radicals.